Articles
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Carpal hyperextension is an abnormality of the carpus that causes increased extension of the joint. There are many causes of carpal hyperextension: it can be caused by trauma; in young dogs it may be caused by a developmental abnormality; and in older dogs it may occur as a degenerative condition. Dogs with carpal hyperextension have a noticeable bend at the wrist, forcing their lower limb into an abnormally flattened position. If carpal hyperextension is caused by trauma, it may also be associated with pain and swelling. A tentative diagnosis of carpal hyperextension can be made based on initial observation, but a thorough physical examination is necessary because dogs with carpal hyperextension may also have abnormalities in other joints. Treatment of carpal hyperextension depends upon the cause and severity of the condition and can range from medical management to surgery.
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Carpal laxity is a condition in which the carpus has an abnormal or excessive range of motion. Carpal laxity can show up in one of two ways: carpal hyperextension or carpal flexion. The underlying cause of carpal laxity has not been definitively determined but may be caused by nutritional factors (specifically excessive caloric intake and/or excessive calcium intake), genetic factors, and being raised on slippery flooring surfaces. Signs of carpal laxity may be seen at any time from 6 weeks of age onward, but the condition is most commonly noted between three and six months of age. Activity modification is often recommended for affected puppies by keeping them off slippery surfaces. Most puppies with carpal laxity will appear completely normal within six to eight weeks.
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The intervertebral discs allow movement in the spine and act as shock absorbers between the vertebrae. If the disc degenerates or is damaged in some way, the disc may bulge and put pressure on the spinal cord and/or the roots of the spinal nerves that come off the sides of the spinal cord. This pressure can cause symptoms ranging from severe pain to weakness to paralysis. There are several breeds that experience a higher frequency of the condition. The severity of a dog’s clinical signs depends upon several factors. Conservative management with pain relievers and anti-inflammatory medication is recommended with a gradual onset of clinical signs or when clinical signs are limited to pain and/or a mildly wobbly gait. Surgery is recommended when there are repeated episodes of neck pain, when neck pain is severe, when there are severe nervous system deficits, or when the dog has not responded to conservative treatment.
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This handout summarizes the normal function of the dog heart, and outlines the abnormalities and changes associated with degeneration of the four different valves found in the heart. An explanation is provided of clinical signs associated with valve disease, as well as the recommended tests and treatments for this condition. Staging of heart disease is also discussed.
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Chronic lymphocytic leukemia is a slowly progressing cancer of a type of white blood cell called a lymphocyte. It can develop at any age but is more typically detected in middle-aged to senior dogs. It also seems to be more prevalent in German Shepherds and Golden Retrievers. This disease is often asymptomatic and detected on routine lab screening. Further diagnostic procedures, as well as treatments and prognoses, are described in this handout.
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Clostridial enterotoxicosis is a complex and poorly understood syndrome characterized by diarrhea that is associated with the bacteria Clostridium perfringens (CP). It is associated with either acute or chronic diarrhea. Clostridial enterotoxicosis is not caused by the bacteria directly, but rather by a toxin produced by the bacteria. Most cases of intestinal bacterial overgrowth are treated on an outpatient basis using antibiotics, nutrition, and probiotics.
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Coccidioidomycosis is a fungal disease caused by the soil fungus Coccidioides immitis or Coccidioides posadii. The early signs of coccidioidomycosis include fever, lethargy, lack of appetite, coughing, and joint pain. Diagnosis is by biopsy of lesions or blood testing for antibodies to the Coccidioides organism. The prognosis for recovery is dependent of the extent of lesions in the dog’s body.
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Collie Eye Anomaly (CEA) is a genetic mutation affecting many breeds that causes developmental defects in the eye that can lead to vision deficits or blindness. This defect can be diagnosed by a veterinary ophthalmologist between 6 and 8 weeks of age by visualizing spots of choroidal hypoplasia or a colobomas . It can be associated with microphthalmia or enophthalmia. It can lead to retinal detachment and blindness. Although laser repair of partial retinal detachments can be attempted if detected in early stages, there is generally no treatment for CEA. Vision varies depending on the extent of the lesions and some dogs will become blind. Prevention requires not breeding animals that carry the mutation and this can be achieved through genetic testing of breeding dogs.
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Color dilution alopecia is an autosomal recessive genetic disorder that affects dogs with dilute-colored coats causing hair loss. Some dogs will also be affected by secondary bacterial infections so topical management and pruritus treatments may be needed to improve the dog's comfort. Affected dogs and their relatives should not be bred.
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Coonhound paralysis is a sudden inflammation of multiple nerve roots and peripheral nerves in dogs, and occasionally cats. It can be caused by an immune reaction to raccoon saliva. It can also occur in dogs who have not encountered a raccoon. In this case, it is called acute idiopathic polyradiculoneuritis, and its cause is often unknown. Dogs with coonhound paralysis start out with a stiff-legged gait that rapidly progresses to paralysis of all four legs. Good nursing care is essential for recovery.
